Prevention against Coronavirus and how to protect yourself?

Strong immune system to fight off the Coronavirus

Prevention against coronavirus immune system

Prevention of the coronavirus can be understood if we look at what the coronavirus really is.

Many of those affected by coronavirus develop only a mild illness, while some develop no illness at all. That may be an indication of what happens when the virus meets a well-functioning immune system. Antioxidants such as Organic Lycopene can help.

When it comes to keeping our immune system healthy it’s important to focus on healthy habits. When your immune system is strong you are more prepared to fight viruses off like the coronavirus; Most are sailing through the epidemic just fine, a strong immune system is one of the best prevention methods against the coronavirus.

Previous suggested advice to prevent the common flu this season is to eat well, sleep well and de-stress. This will help combat the corona virus (COVID-19)

For most healthy individuals the flu is more of an inconvenience than something that can be fatal or life-threatening.

Doctors still recommend adopting healthy habits that can keep the immune system in great form, like staying well-hydrated and eating a balanced diet.

What’s even more crucial is reducing stress, which can directly affect immune function and be a prevention against coronavirus.

Excess amounts of stress can suppress your immune response to bacteria and viruses.

“We’re assaulted on a daily basis from many different  viruses coming from our kids, our colleagues, and surfaces that we touch throughout the day”

Exercise is beneficial for a healthy immune system as it benefits just about everything along with the right amount of sleep as it’s important to keep the immune system functioning optimal.

How to protect yourself: Prevention against coronavirus

Boost Immune System with organic LycopeneBoost your Immune System

Boost your Immune System. Use Organic Lycopene Supplement to fight free radicals. The human lung, due to the oxidative and ozone stress to which it is exposed, is particularly vulnerable to oxidative damage. Concentrations of dietary antioxidants in the lung epithelial lining and lining fluids may provide protection against oxidative damage.

get enough sleep helps with prevention against coronavirus

Get enough sleep.

It really does work. Immune system signaling chemicals called cytokines are involved in sleep, and people who don’t get enough sleep are not only more prone to infections, but they do not get a good response to vaccines, either. Research has suggested that each night, adults need at least seven to eight hours of sleep.

eat-healthly to boos immune systemEat enough fruits, vegetables and whole grains.

Fruits and vegetables provide the obvious vitamins, including A, C and E, but also other nutrients and antioxidants.Studies show a huge range of health benefits from eating a variety of fruits and vegetables, there’s not much evidence to show any particular fruit or product bestows special immunity. The key is variety, number and color — colorful fruits and vegetables tend to contain more vitamins and antioxidants such as lycopene (Organic Lycopene Supplement, one capsule is equal to eating 6lbs of tomatoes) and lutein. Fighting free radicals may help to keep your immune system strong and is a prevention against COVID-19

Drink moderate

Drink moderately

A recent study found that people who downed the equivalent of four or five drinks in one session showed suppressed immune systems for several hours afterwards. Other studies show that people who chronically drink too much weaken their immune systems over time. People who abuse alcohol are more vulnerable to pneumonia, septicemia and hepatitis C and have a weak prevention against coronavirus.

don't smokeDon’t smoke.

You can add a weakened immune system to the very long list of ills caused by smoking. Nicotine — found in traditional cigarettes and in e-cigarettes — skews the function of immune cells called macrophages. Various hydrocarbons found in cigarette smoke damage many different immune cells, including lymphocytes and macrophages.

Start moving.

Many studies show people who get regular, moderate exercise are less likely to catch a range of infections, including colds and flu. Exercise helps with the prevention of coronavirus.


Protection from the Coronavirus

Who is getting sick from the coronavirus, and how sick?

The new coronavirus is not an equal-opportunity killer: Being elderly and having other illnesses, for instance, greatly increases the risk of dying from the disease 

the virus causes, Covid-19. It’s also possible that being male could put you at an increased risk.

Much is still unknown about the coronavirus which started in China, but one thing is certain. The disease can cast a storm over the whole human body.

Such has been the nature of past zoonotic coronaviruses, ones that hopped from animals to humans like SARS and MERS. Unlike their common-cold-causing cousins, these emergent coronaviruses can spark a viral-induced fire throughout many of a person’s organs, and the new disease—dubbed “COVID-19” by the World Health Organization—is no exception when it is severe.

That helps explain why the COVID-19 epidemic has killed more than 1,800 people, surpassing the SARS death toll in a matter of weeks. While the death rate for COVID-19 appears to be a fifth of SARS, the coronavirus has spread faster.

Confirmed cases rose to more than 60,000 last Thursday, nearly a 50 percent jump relative to the prior day, and the tally has since increased by another 13,000. This leap reflects a change in the way Chinese authorities are diagnosing infections instead of a massive shift in the scope of the outbreak. Rather than wait for patients to test positive for the virus, diagnoses now include anyone whose chest scan reveals COVID-19’s distinctive pattern of pneumonia. This method will hopefully allow authorities to isolate and treat patients more quickly.

If this outbreak continues to spread, there’s no telling how harmful it could become. A leading epidemiologist at the University of Hong Kong warned this week that COVID-19 could infect 60 percent of the globe if left unchecked.

On Monday, the Chinese Center for Disease Control and Prevention against Coronavirus released clinical details on the first 72,314 patients diagnosed through February 11. The report shows that COVID-19 killed 2.3 percent of patients, meaning it is currently 23 times more fatal than the seasonal flu. Severe disease and deaths were reported in every age group, except kids under nine years old.

Meanwhile, countries have been scrambling to evacuate their citizens from a cruise ship that’s been quarantined off the coast of Japan. The Japanese health ministry announced Tuesday that an additional 88 passengers on the ship were diagnosed with COVID-19—bringing the total to 542. Of the more than 300 Americans who have been repatriated from the ship, 14 have tested positive for the coronavirus.

Old and young

The vast majority of cases in China — 87% — were in people ages 30 to 79, the China Center for Disease Control reported last month based on data from all 72,314 of those diagnosed with Covid-19 as of Feb. 11.

This shows something about biology more than lifestyle, such as being in frequent contact with other people.

Teens and people in their 20s also encounter many others, at school and work and on public transit, yet they don’t seem to be contracting the disease at significant rates: Only 8.1% of cases were 20-somethings, 1.2% were teens, and 0.9% were 9 or younger. The World Health Organization mission to China found that 78% of the cases reported as of Feb. 20 were in people ages 30 to 69.

The death toll skews old even more strongly. Overall, China CDC found, 2.3% of confirmed cases died. But the fatality rate was 14.8% in people 80 or older, likely reflecting the presence of other diseases, a weaker immune system, or simply worse overall health. By contrast, the fatality rate was 1.3% in 50-somethings, 0.4% in 40-somethings, and 0.2% in people 10 to 39.

The age-related death risk probably reflects the strength, or weakness, of the respiratory system. About half of the 109 Covid-19 patients (ages 22 to 94) treated at Central Hospital of Wuhan, researchers there reported, developed acute respiratory distress syndrome (ARDS), in which fluid builds up in the small air sacs of the lungs. That restricts how much air the lungs can take in, reducing the oxygen supply to vital organs, sometimes fatally; half of the ARDS patients died, compared to 9% of patients who did not develop the syndrome.

The ARDS patients had an average age of 61, compared to an average age of 49 for those who did not develop ARDS. Elderly patients “were more likely to develop ARDS,” the researchers wrote, suggesting how age can make Covid-19 more severe and even fatal: age increases the risk that the respiratory system will basically shut down under viral assault.

It is important for the old to take extra prevention against coronavirus and a Organic Lycopene supplement that helps with boosting the immune system is wise. 

Sick or healthy: they need more prevention against coronavirus.

The male-female difference in fatality rates, and perhaps in reported incidence, may arise from differences in underlying health. People with pre-existing illness are more likely to get seriously ill from Covid-19, and men have a higher incidence of such chronic illnesses as cardiovascular disease.

In the first large study of the effect of underlying illness, researchers in China analyzed 1,590 patients from throughout the country with laboratory-confirmed disease. They calculated how “co-morbidities” — existing illnesses — affected the risk of being admitted to intensive care, being put on a ventilator, or dying.

After taking into account the patients’ ages and smoking status, the researchers found that the 399 patients with at least one additional disease (including cardiovascular diseases, diabetes, hepatitis B, chronic obstructive pulmonary disease, chronic kidney diseases, and cancer) had a 79% greater chance of requiring intensive care or a respirator or both, or of dying, they reported last week in a paper posted to medRxiv, a preprint site that posts research before it has been peer-reviewed. The 130 with two or more additional diseases had 2.5 times the risk of any of those outcomes.

That fits with what’s known about other respiratory viruses. People with a single co-morbidity who catch avian flu (H7N9) are 3.4 times more likely than otherwise-healthy flu patients to require ventilators and other intensive care. SARS and MERS, which are also caused by coronaviruses, are more severe in patients with underlying illnesses, too.

Breaking down the Covid-19 risk with the most common co-morbidities, the scientists found that cancer raises the risk 3.5-fold, COPD 2.6-fold, and diabetes and hypertension by about 60%. Because it isn’t unusual for someone to have an underlying disease (especially diabetes and hypertension) but not know it, the last figure is likely an underestimate.

Co-morbidities also raise the risk of dying from Covid-19. China CDC’s analysis of 44,672 patients found that the fatality rate in patients who reported no other health conditions was 0.9%. It was 10.5% for those with cardiovascular disease, 7.3% for those with diabetes, 6.3% for people with chronic respiratory diseases such as COPD, 6.0% for people with hypertension, and 5.6% for those with cancer.

Underlying disease might change the course of Covid-19. During the height of the epidemic in Wuhan, 37 of 230 patients receiving dialysis for kidney failure at Remnin Hospital developed the disease. Even though none were sick enough to require intensive care or a mechanical ventilator, six of them died, for a very high fatality rate of 16%. Oddly, however, none of the six died of pneumonia, Remnin researchers reported. Instead, the causes of death were heart disease, stroke, and high blood levels of potassium (a result of kidney failure). The high fatality rate of Covid-19 in already-sick people might result not from the virus but from an exacerbation of existing disease.

About 60% of U.S. adults have at least one underlying health condition, Tom Frieden, former director of the U.S. Centers for Disease Control and Prevention, told reporters on Monday.

But what actually happens to your body when it is infected by the coronavirus?

For most patients, COVID-19 begins and ends in their lungs, because like the flu, coronaviruses are respiratory diseases.

They spread typically when an infected person coughs or sneezes, spraying droplets that can transmit the virus to anyone in close contact. Coronaviruses also cause flu-like symptoms: Patients might start out with a fever and cough that progresses to pneumonia or worse. Prevention against coronavirus is like fighting the flu.

After the SARS outbreak, the WHO reported that the disease attacked the lungs in three phases:

  • viral replication
  • immune hyper-reactivity
  • and pulmonary destruction.

In the early days of an infection, the coronavirus rapidly invades human lung cells. Those lung cells come in two classes:

  • ones that make mucus
  • ones with hair-like batons called cilia

Phase One: Mucus, though gross when outside the body, helps protect lung tissue from pathogens and make sure your breathing organ doesn’t dry out. The cilia cells beat around the mucus, clearing out debris like pollen or viruses.

SARS starts to infect and kill cilia cells, which then sloughed off and filled patients’ airways with debris and fluids, and  many believe that the same happens with the COVID-19

That’s when Phase Two and the immune system kicks in. Aroused by the presence of a viral invader, our bodies step up to fight the disease by flooding the lungs with immune cells to clear away the damage and repair the lung tissue.

When the immune system is working properly, this inflammatory process is tightly regulated and confined only to infected areas. But sometimes it goes off track start to kill anything in their way, including your healthy tissue.

So you get more damage instead of less from the immune response

During the Third Phase, lung damage continues to build—which can result in respiratory failure. Even if death doesn’t occur, some patients survive with permanent lung damage. According to the WHO, SARS punched holes in the lungs, giving them “a honeycomb-like appearance”—and these organ/tissue damages are present in those afflicted by coronavirus, too.

These holes are likely created by the immune system’s hyperactive response, which creates scars that both protect and stiffen the lungs.

When that occurs, patients often have to be put on ventilators to assist their breathing. Meanwhile, inflammation also makes the membranes between the air sacs and blood vessels more permeable, which can fill the lungs with fluid and affect their ability to oxygenate blood.

In severe cases, you basically flood your lungs and you can’t breathe, That’s how people are dying.

Conclusion: keep your immune system optimal prevention against coronavirus

As there is no known cure for it yet, while many patients have recovered from their symptoms as their immune system fought off the COVID-19.

So you can help your immune system by eliminating as much risk as possible to optimize the prevention against coronavirus:

  • Wash your hands often with soap and water for at least 20 seconds. Don’t forget to wash the backs of your hands and under your nails. After going to the bathroom; before eating; and after blowing your nose, coughing, or sneezing.
  • Avoid touching your eyes, nose, and mouth.
  •  Stay at least 3 feet (or 1 meter) away from anyone who may be infected.
  • If you’re the one feeling sick, cover your entire mouth and nose when you cough or sneeze with a tissue, then throw the tissue in the trash. But don’t use your hands.
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